| Rapamycin-induced G1 cell cycle arrest employs both TGF-β and Rb pathways A Chatterjee, S Mukhopadhyay, K Tung, D Patel, DA Foster Cancer letters 360 (2), 134-140, 2015 | 70 | 2015 |
| Blocking anaplerotic entry of glutamine into the TCA cycle sensitizes K-Ras mutant cancer cells to cytotoxic drugs M Saqcena, S Mukhopadhyay, C Hosny, A Alhamed, A Chatterjee, ... Oncogene 34 (20), 2672-2680, 2015 | 63 | 2015 |
| Reciprocal regulation of AMP-activated protein kinase and phospholipase D S Mukhopadhyay, M Saqcena, A Chatterjee, A Garcia, MA Frias, ... Journal of Biological Chemistry 290 (11), 6986-6993, 2015 | 58 | 2015 |
| 5-Aminoimidazole-4-carboxamide-1-β-4-ribofuranoside (AICAR) enhances the efficacy of rapamycin in human cancer cells S Mukhopadhyay, A Chatterjee, D Kogan, D Patel, DA Foster Cell Cycle, 00-00, 2015 | 43 | 2015 |
| Inhibition of S6 kinase suppresses the apoptotic effect of eIF4E ablation by inducing TGF-β-dependent G1 cell cycle arrest P Yellen, A Chatterjee, A Preda, DA Foster Cancer letters 333 (2), 239-243, 2013 | 25 | 2013 |
| Control of cell cycle progression by mTOR A Chatterjee CITY UNIVERSITY OF NEW YORK, 2015 | 2 | 2015 |
| Cytotoxic and cystostatic properties of rapamycin are due to differential effects on the phosphorylation of mTORC1 substrates S6 kinase and 4E-BP1 P Yellen, A Chatterjee, M Saqcena, D Salloum, A Preda, ... Cancer Research 72 (8 Supplement), 2798-2798, 2012 | | 2012 |
