|Coronary plaque disruption|
E Falk, PK Shah, V Fuster
Circulation 92 (3), 657-671, 1995
|ESC Guidelines for the management of acute coronary syndromes in patients presenting without persistent ST-segment elevation: The Task Force for the management of acute …|
Authors/Task Force Members, CW Hamm, JP Bassand, S Agewall, J Bax, ...
European heart journal 32 (23), 2999-3054, 2011
|From vulnerable plaque to vulnerable patient: a call for new definitions and risk assessment strategies: Part I|
M Naghavi, P Libby, E Falk, SW Casscells, S Litovsky, J Rumberger, ...
Circulation 108 (14), 1664-1672, 2003
|Management of acute myocardial infarction in patients presenting with ST-segment elevation|
F Van de Werf, D Ardissino, A Betriu, DV Cokkinos, E Falk, KAA Fox, ...
European heart journal 24 (1), 28-66, 2003
|Mechanisms of plaque formation and rupture|
JF Bentzon, F Otsuka, R Virmani, E Falk
Circulation research 114 (12), 1852-1866, 2014
|Macrophage infiltration in acute coronary syndromes. Implications for plaque rupture.|
PR Moreno, E Falk, IF Palacios, JB Newell, V Fuster, JT Fallon
Circulation 90 (2), 775-778, 1994
|Pathogenesis of atherosclerosis|
Journal of the American College of cardiology 47 (8S), C7-C12, 2006
|Unstable angina with fatal outcome: dynamic coronary thrombosis leading to infarction and/or sudden death. Autopsy evidence of recurrent mural thrombosis with peripheral …|
Circulation 71 (4), 699-708, 1985
|Plaque rupture with severe pre-existing stenosis precipitating coronary thrombosis. Characteristics of coronary atherosclerotic plaques underlying fatal occlusive thrombi.|
Heart 50 (2), 127-134, 1983
|Update on acute coronary syndromes: the pathologists' view|
E Falk, M Nakano, JF Bentzon, AV Finn, R Virmani
European heart journal 34 (10), 719-728, 2013
|Human monocyte-derived macrophages induce collagen breakdown in fibrous caps of atherosclerotic plaques. Potential role of matrix-degrading metalloproteinases and implications …|
PK Shah, E Falk, JJ Badimon, A Fernandez-Ortiz, A Mailhac, ...
Circulation 92 (6), 1565-1569, 1995
|Why do plaques rupture?|
Circulation 86 (6 Suppl), III30-42, 1992
|From vulnerable plaque to vulnerable patient—part III: executive summary of the Screening for Heart Attack Prevention and Education (SHAPE) Task Force report|
M Naghavi, E Falk, HS Hecht, MJ Jamieson, S Kaul, D Berman, Z Fayad, ...
The American journal of cardiology 98 (2), 2-15, 2006
|Characterization of the relative thrombogenicity of atherosclerotic plaque components: implications for consequences of plaque rupture|
A Fernández-Ortiz, JJ Badimon, E Falk, V Fuster, B Meyer, A Mailhac, ...
Journal of the American College of Cardiology 23 (7), 1562-1569, 1994
|Terminology for high-risk and vulnerable coronary artery plaques|
JA Schaar, JE Muller, E Falk, R Virmani, V Fuster, PW Serruys, ...
European heart journal 25 (12), 1077-1082, 2004
|Morphologic features of unstable atherothrombotic plaques underlying acute coronary syndromes|
The American journal of cardiology 63 (10), E114-E120, 1989
|Prevalence, impact, and predictive value of detecting subclinical coronary and carotid atherosclerosis in asymptomatic adults: the BioImage study|
U Baber, R Mehran, S Sartori, MM Schoos, H Sillesen, P Muntendam, ...
Journal of the American College of Cardiology 65 (11), 1065-1074, 2015
|In vivo heating of pacemaker leads during magnetic resonance imaging|
R Luechinger, VA Zeijlemaker, EM Pedersen, P Mortensen, E Falk, ...
European heart journal 26 (4), 376-383, 2005
|Right ventricular infarction: frequency, size and topography in coronary heart disease: a prospective study comprising 107 consecutive autopsies from a coronary care unit|
HR Andersn, E Falk, D Nielsen
Journal of the American College of Cardiology 10 (6), 1223-1232, 1987
|Recommendation on design, execution, and reporting of animal atherosclerosis studies: a scientific statement from the American Heart Association|
A Daugherty, AR Tall, MJAP Daemen, E Falk, EA Fisher, ...
Arteriosclerosis, thrombosis, and vascular biology 37 (9), e131-e157, 2017