|Cellular basis of abnormal calcium transients of failing human ventricular myocytes|
V Piacentino III, CR Weber, X Chen, J Weisser-Thomas, KB Margulies, ...
Circulation research 92 (6), 651-658, 2003
|Ca2+- and mitochondrial-dependent cardiomyocyte necrosis as a primary mediator of heart failure|
H Nakayama, X Chen, CP Baines, R Klevitsky, X Zhang, H Zhang, ...
The Journal of clinical investigation 117 (9), 2431-2444, 2007
|Bone marrow cells adopt the cardiomyogenic fate in vivo|
M Rota, J Kajstura, T Hosoda, C Bearzi, S Vitale, G Esposito, G Iaffaldano, ...
Proceedings of the National Academy of Sciences 104 (45), 17783-17788, 2007
|A peptide encoded by a transcript annotated as long noncoding RNA enhances SERCA activity in muscle|
BR Nelson, CA Makarewich, DM Anderson, BR Winders, CD Troupes, ...
Science 351 (6270), 271-275, 2016
|L-Type Ca2+ Channel Density and Regulation Are Altered in Failing Human Ventricular Myocytes and Recover After Support With Mechanical Assist Devices|
X Chen, V Piacentino III, S Furukawa, B Goldman, KB Margulies, ...
Circulation research 91 (6), 517-524, 2002
|Ca2+ Influx–Induced Sarcoplasmic Reticulum Ca2+ Overload Causes Mitochondrial-Dependent Apoptosis in Ventricular Myocytes|
X Chen, X Zhang, H Kubo, DM Harris, GD Mills, J Moyer, R Berretta, ...
Circulation research 97 (10), 1009-1017, 2005
|G protein-coupled receptor kinase 2 ablation in cardiac myocytes before or after myocardial infarction prevents heart failure|
PW Raake, LE Vinge, E Gao, M Boucher, G Rengo, X Chen, ...
Circ Res 103, 413-422, 2008
|CaMKII negatively regulates calcineurin–NFAT signaling in cardiac myocytes|
SM MacDonnell, J Weisser-Thomas, H Kubo, M Hanscome, Q Liu, ...
Circulation research 105 (4), 316-325, 2009
|Adolescent feline heart contains a population of small, proliferative ventricular myocytes with immature physiological properties|
X Chen, RM Wilson, H Kubo, RM Berretta, DM Harris, X Zhang, N Jaleel, ...
Circulation research 100 (4), 536-544, 2007
|Protein kinase Cα, but not PKCβ or PKCγ, regulates contractility and heart failure susceptibility: implications for ruboxistaurin as a novel therapeutic approach|
Q Liu, X Chen, SM MacDonnell, EG Kranias, JN Lorenz, M Leitges, ...
Circulation research 105 (2), 194-200, 2009
|Decreased cardiac L-type Ca2+ channel activity induces hypertrophy and heart failure in mice|
SA Goonasekera, K Hammer, M Auger-Messier, I Bodi, X Chen, H Zhang, ...
The Journal of clinical investigation 122 (1), 280-290, 2012
|GDF11 does not rescue aging-related pathological hypertrophy|
SC Smith, X Zhang, X Zhang, P Gross, T Starosta, S Mohsin, M Franti, ...
Circulation research 117 (11), 926-932, 2015
|Cardiotoxic and Cardioprotective Features of Chronic β-Adrenergic SignalingNovelty and Significance|
X Zhang, C Szeto, E Gao, M Tang, J Jin, Q Fu, C Makarewich, X Ai, Y Li, ...
Circulation Research 112 (3), 498-509, 2013
|Repair of the injured adult heart involves new myocytes potentially derived from resident cardiac stem cells|
D Angert, RM Berretta, H Kubo, H Zhang, X Chen, W Wang, B Ogorek, ...
Circulation research 108 (10), 1226-1237, 2011
|L-Type Ca 2+ Channel α1cSubunit Isoform Switching in Failing Human Ventricular Myocardium|
Y Yang, X Chen, K Margulies, V Jeevanandam, P Pollack, BA Bailey, ...
Journal of molecular and cellular cardiology 32 (6), 973-984, 2000
|Adrenergic regulation of cardiac contractility does not involve phosphorylation of the cardiac ryanodine receptor at serine 2808|
SM MacDonnell, G García-Rivas, JA Scherman, H Kubo, X Chen, ...
Circulation research 102 (8), e65-e72, 2008
|The mitochondrial Na+/Ca2+ exchanger is essential for Ca2+ homeostasis and viability.|
TS Luongo, JP Lambert, P Gross, M Nwokedi, AA Lombardi, ...
|α1G-dependent T-type Ca2+ current antagonizes cardiac hypertrophy through a NOS3-dependent mechanism in mice|
H Nakayama, I Bodi, RN Correll, X Chen, J Lorenz, SR Houser, J Robbins, ...
The Journal of clinical investigation 119 (12), 3787-3796, 2009
|Hyperphosphorylation of the cardiac ryanodine receptor at serine 2808 is not involved in cardiac dysfunction after myocardial infarction|
H Zhang, CA Makarewich, H Kubo, W Wang, JM Duran, Y Li, RM Berretta, ...
Circulation research 110 (6), 831-840, 2012
|Calcium influx through Cav1. 2 is a proximal signal for pathological cardiomyocyte hypertrophy|
X Chen, H Nakayama, X Zhang, X Ai, DM Harris, M Tang, H Zhang, ...
Journal of molecular and cellular cardiology 50 (3), 460-470, 2011