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Matthew Utter
Matthew Utter
Weill Cornell Medicine
Verified email at med.cornell.edu
Title
Cited by
Cited by
Year
Phosphatidic acid drives mTORC1 lysosomal translocation in the absence of amino acids
MA Frias, S Mukhopadhyay, E Lehman, A Walasek, M Utter, D Menon, ...
Journal of Biological Chemistry 295 (1), 263-274, 2020
282020
Suppression of AKT phosphorylation restores rapamycin-based synthetic lethality in SMAD4-defective pancreatic cancer cells
O Le Gendre, A Sookdeo, SA Duliepre, M Utter, M Frias, DA Foster
Molecular Cancer Research 11 (5), 474-481, 2013
252013
Elevated phospholipase D activity in androgen-insensitive prostate cancer cells promotes both survival and metastatic phenotypes
M Utter, S Chakraborty, L Goren, L Feuser, YS Zhu, DA Foster
Cancer letters 423, 28-35, 2018
142018
Cancer cells with defective RB and CDKN2A are resistant to the apoptotic effects of rapamycin
S Chakraborty, MB Utter, MA Frias, DA Foster
Cancer letters 522, 164-170, 2021
92021
RB and CDKN2A cause Rapamycin resistance in cancer cells
S Chakraborty, M Utter, D Foster
The FASEB Journal 33 (S1), lb283-lb283, 2019
2019
Mechanisms for Survival and Drug Resistance in Cancer Cells
M Utter
City University of New York, 2018
2018
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Articles 1–6